Dengue virus neither directly mediates hyperpermeability nor enhances tumor necrosis factor-α-induced permeability in vitro.

@article{Raekiansyah2014DengueVN,
  title={Dengue virus neither directly mediates hyperpermeability nor enhances tumor necrosis factor-α-induced permeability in vitro.},
  author={Muhareva Raekiansyah and Lyre Anni Espada-Murao and Kenta Okamoto and Toru Kubo and Kouichi Morita},
  journal={Japanese journal of infectious diseases},
  year={2014},
  volume={67 2},
  pages={86-94}
}
The mechanisms of endothelial barrier dysfunction in dengue disease remain poorly understood. Endothelial cell (EC) death due to virus infection or in combination with an infection-induced cytokine storm is deemed as one of the major causes of plasma leakage. Using an in vitro model of human endothelia and several dengue virus (DENV) strains (including a clinical isolate), the direct consequence of infection on endothelial permeability was investigated throughout the course of the infection… CONTINUE READING
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