Deficiency of intramuscular ICC increases fundic muscle excitability but does not impede nitrergic innervation.

@article{Huizinga2008DeficiencyOI,
  title={Deficiency of intramuscular ICC increases fundic muscle excitability but does not impede nitrergic innervation.},
  author={Jan D. Huizinga and Louis W C Liu and Amanda Fitzpatrick and Elizabeth H. White and Sandeep Gill and Xuan-Yu Wang and Natalia Z{\'a}rate and Lynn Krebs and Catherine J Choi and Tanya Starret and Devika Dixit and Jing Ye},
  journal={American journal of physiology. Gastrointestinal and liver physiology},
  year={2008},
  volume={294 2},
  pages={
          G589-94
        }
}
The motility of the gastrointestinal tract is generated by smooth muscle cells and is controlled to a large extent by an intrinsic neural network. A gap of approximately 200 nm usually separates nerve varicosities from smooth muscle cells, which suggests that direct innervation of the smooth muscle by synapses does not occur. Enteric nerves do make synapse-like contact with proposed regulatory cells, the interstitial cells of Cajal (ICC), which in turn may be in gap junction contact with smooth… CONTINUE READING

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