Cutaneous neuronal nitric oxide is specifically decreased in postural tachycardia syndrome.

@article{Stewart2007CutaneousNN,
  title={Cutaneous neuronal nitric oxide is specifically decreased in postural tachycardia syndrome.},
  author={Julian M. Stewart and Marvin S. Medow and Christopher T Minson and Indu Taneja},
  journal={American journal of physiology. Heart and circulatory physiology},
  year={2007},
  volume={293 4},
  pages={H2161-7}
}
Low flow postural tachycardia syndrome (POTS), is associated with reduced nitric oxide (NO) activity assumed to be of endothelial origin. We tested the hypothesis that cutaneous microvascular neuronal NO (nNO) is impaired, rather than endothelial NO (eNO), in POTS. We performed three sets of experiments on subjects aged 22.5 +/- 2 yr. We used laser-Doppler flowmetry response to sequentially increase acetylcholine (ACh) doses and the local cutaneous heating response of the calf as bioassays for… CONTINUE READING
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