Correction: Pristane-Induced Arthritis Loci Interact with the Slc11a1 Gene to Determine Susceptibility in Mice Selected for High Inflammation

@inproceedings{Franco2014CorrectionPA,
  title={Correction: Pristane-Induced Arthritis Loci Interact with the Slc11a1 Gene to Determine Susceptibility in Mice Selected for High Inflammation},
  author={Marcelo Fabiano de Franco and Luciana Carla Peters and Mara Adriana Correa and A. Pacheco Galv{\'a}n and Tatiane Canhamero and Andrea Borrego and Jos{\'e} Ricardo Jensen and Jussara Maria Gonçalves and Wafa Hanna Koury Cabrera and Nancy Starobinas and Orlando Garcia Ribeiro and Tommaso A Dragani and Olga C{\'e}lia Martinez Iba{\~n}ez},
  booktitle={PloS one},
  year={2014}
}
AIRmax (maximal inflammation) and AIRmin (minimal inflammation) mice show distinct susceptibilities to pristane-induced arthritis (PIA). The Slc11a1 gene, which regulates macrophage and neutrophil activity, is involved in this infirmity. AIRmax (SS) mice homozygous for the non-functional Slc11a1 S (gly169asp) allele obtained by genotype-assisted crosses from AIRmax and AIRmin mice are more susceptible than mice homozygous for the Slc11a1 resistant (R) allele. The present work sought to identify… CONTINUE READING