Core2 1-6-N-Glucosaminyltransferase-I Deficiency Protects Injured Arteries From Neointima Formation in ApoE-Deficient Mice

@article{Wang2009Core21D,
  title={Core2 1-6-N-Glucosaminyltransferase-I Deficiency Protects Injured Arteries From Neointima Formation in ApoE-Deficient Mice},
  author={Huan Wang and Weiyu Zhang and Rong Tang and R. Hebbel and M. Kowalska and C. Zhang and J. Marth and M. Fukuda and Chuhong Zhu and Y. Huo},
  journal={Arteriosclerosis, Thrombosis, and Vascular Biology},
  year={2009},
  volume={29},
  pages={1053-1059}
}
Objective—Core2 1 to 6-N-glucosaminyltransferase-I (C2GlcNAcT-I) plays an important role in optimizing the binding functions of several selectin ligands, including P-selectin glycoprotein ligand. We used apolipoprotein E (ApoE)-deficient atherosclerotic mice to investigate the role of C2GlcNAcT-I in platelet and leukocyte interactions with injured arterial walls, in endothelial regeneration at injured sites, and in the formation of arterial neointima. Methods and Results—Arterial neointima… Expand
Adenosine Receptor A2A Deficiency in Leukocytes Increases Arterial Neointima Formation in Apolipoprotein E–Deficient Mice
P-selectin glycoprotein ligand-1 plays a crucial role in the selective recruitment of leukocytes into the atherosclerotic arterial wall.
  • Y. Huo, L. Xia
  • Biology, Medicine
  • Trends in cardiovascular medicine
  • 2009
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Core2 1-6-N-Glucosaminyltransferase-I Is Crucial for the Formation of Atherosclerotic Lesions in Apolipoprotein E–Deficient Mice
Critical Role of Platelet P-Selectin in the Response to Arterial Injury in Apolipoprotein-E–Deficient Mice
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