Complement receptor 2 polymorphisms associated with systemic lupus erythematosus modulate alternative splicing

@inproceedings{Douglas2009ComplementR2,
  title={Complement receptor 2 polymorphisms associated with systemic lupus erythematosus modulate alternative splicing},
  author={Katherine B. Douglas and Daniel C. Windels and Jian Zhao and Agnessa V. Gadeliya and Hui Wu and Kenneth M Kaufman and John Barker Harley and Joan Tenenbaum Merrill and Robert P. Kimberly and Graciela S Alarc{\'o}n and Elizabeth E. Brown and Jeffrey C. Edberg and Rosalind Ramsey-Goldman and Michelle Petri and John D Reveille and Luis M Vil{\'a} and Patrick M Gaffney and Judith A. James and Kathy L. Moser and Marta E. Alarc{\'o}n-Riquelme and Timothy J Vyse and Gary S. Gilkeson and Chaim O. Jacob and Julie T. Ziegler and Carl D. Langefeld and Daniela Ulgiati and Betty P. Tsao and Susan A. Boackle},
  booktitle={Genes and Immunity},
  year={2009}
}
Genetic factors influence susceptibility to systemic lupus erythematosus (SLE). A recent family-based analysis in Caucasian and Chinese populations provided evidence for association of single-nucleotide polymorphisms (SNPs) in the complement receptor 2 (CR2/CD21) gene with SLE. Here we confirmed this result in a case–control analysis of an independent… CONTINUE READING