Co-expression/co-location of S100 proteins (S100B, S100A1 and S100A2) and protein kinase C (PKC-beta, -eta and -zeta) in a rat model of cerebral basilar artery vasospasm.

@article{Lefranc2005CoexpressioncolocationOS,
  title={Co-expression/co-location of S100 proteins (S100B, S100A1 and S100A2) and protein kinase C (PKC-beta, -eta and -zeta) in a rat model of cerebral basilar artery vasospasm.},
  author={Florence Lefranc and Christine Decaestecker and Jacques Brotchi and Claus Wilhelm Heizmann and Olivier Dewitte and R{\'o}bert Kiss and Tatjana Mijatovic},
  journal={Neuropathology and applied neurobiology},
  year={2005},
  volume={31 6},
  pages={649-60}
}
OBJECT The cellular events leading to cerebral vasospasm after subarachnoid haemorrhages (SAH) involve a number of members of the protein kinase C (PKC) family. However, whereas calcium is thought to play a number of major roles in the pathophysiology of SAH, a number of PKCs function independently of calcium. We recently emphasized the potential role of the calcium-binding S100 proteins in a 'double haemorrhage' rat model of SAH-induced vasospasm. A number of S100 proteins are known to… CONTINUE READING