Clinical neurophysiology of postural tachycardia syndrome.

@article{CutsforthGregory2019ClinicalNO,
  title={Clinical neurophysiology of postural tachycardia syndrome.},
  author={Jeremy K. Cutsforth-Gregory and Paola Sandroni},
  journal={Handbook of clinical neurology},
  year={2019},
  volume={161},
  pages={
          429-445
        }
}
Postural Orthostatic Tachycardia Syndrome (POTS): A critical assessment.
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TLDR
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Erratum to “A review of respiratory manifestations and their management in Ehlers-Danlos syndromes and hypermobility spectrum disorders”
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The correct list of references for this article can be found below: 1.
RNA sequencing on muscle biopsy from a 5-week bedrest study reveals the effect of exercise and potential interactions with dorsal root ganglion neurons
TLDR
It is concluded that exercise intervention protects muscle from deconditioning transcriptomic changes, and prevents changes in expression of ligands that might sensitize DRG neurons that promote pain.
RNA sequencing on muscle biopsy from a 5‐week bed rest study reveals the effect of exercise and potential interactions with dorsal root ganglion neurons
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It is concluded that exercise intervention protects muscle from deconditioning transcriptomic changes, and prevents changes in the expression of ligands that might sensitize DRG neurons, or act on other cell types throughout the body.
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References

SHOWING 1-10 OF 150 REFERENCES
Postural tachycardia syndrome—current experience and concepts
TLDR
The characteristics and neuroepidemiology of PoTS are described, and possible pathophysiological mechanisms of this syndrome are outlined, as well as current and investigational treatments.
The Postural Tachycardia Syndrome
TLDR
Therapies for POTS are directed at relieving the central hypovolemia or at compensating for the circulatory dysfunctions that may cause this disorder.
Idiopathic postural orthostatic tachycardia syndrome
TLDR
In many instances POTS may be a manifestation of a mild form of acute autonomic neuropathy, and an excessive orthostatic increase of catecholamines were found in some patients.
The neuropathic postural tachycardia syndrome.
TLDR
The neuropathic postural tachycardia syndrome results from partial sympathetic denervation, especially in the legs, according to the measured norepinephrine spillover.
Postural orthostatic tachycardia syndrome: the Mayo clinic experience.
TLDR
A neuropathic basis for at least half the cases of POTS is suggested and that a substantial percentage of cases may be autoimmune.
Postural tachycardia syndrome: clinical features and follow-up study.
TLDR
In the majority of patients, POTS is self-resolving, especially in those with a triggering event, and salt supplementation and beta-blockers were the most efficacious therapies.
Autonomic dysfunction in fibromyalgia syndrome: Postural orthostatic tachycardia
  • R. Staud
  • Medicine
    Current rheumatology reports
  • 2008
TLDR
Head-up tilt table testing can be used to evaluate autonomic dysfunction and is frequently helpful for the work-up of FM complaints, including fatigue, dizziness, and palpitations.
The value of autonomic testing in postural tachycardia syndrome
TLDR
Clinically, headache and gastrointestinal symptoms were more frequent among the POTS-AN group, suggesting POTS may exist in isolation and may differ from those associated with AN.
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