Chlamydial infections and prostatitis in men

@article{Wagenlehner2006ChlamydialIA,
  title={Chlamydial infections and prostatitis in men},
  author={Florian M. E. Wagenlehner and Kurt G. Naber and Wolfgang Weidner},
  journal={BJU International},
  year={2006},
  volume={97}
}
upon infection into the non-infectious but metabolically active replicating reticulate body (RB) [7]. Within the first 2–6 h after internalization, EBs begin differentiating into RBs. Over the next several hours, RBs increase in number and size, until after 18–24 h the numbers of RBs are maximal [7]. After 24–48 h more RBs begin differentiating back to EBs [7]. The infected cell ruptures at 48–72 h after infection, and the infectious EBs are released again. An intense and chronic inflammation… 
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Although the standard therapy includes intracellularly accumulated antibiotics such as tetracyclines or macrolides, recent evidence favors the role of quinolones, with particular attention to prulifloxacin that is able to decrease IL-8 level and improve the patient's quality of life.
The Role of the Immune Response in Chlamydia trachomatis Infection of the Male Genital Tract: A Double-Edged Sword
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The ability of the immune system to recognize and clear acute and persistent chlamydial infections in the male genital tract is focused on, and on the paradoxical damage that chronic inflammation resulting from the infection can cause on the reproductive health of the individual.
Chlamydia trachomatis infection of the male genital tract: an update.
Re‐emergence of lymphogranuloma venereum
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    Journal of the European Academy of Dermatology and Venereology : JEADV
  • 2008
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LGV is a sexually transmitted infection caused by the L1, L2 and L3 serotypes of Chlamydia trachomatis and most cases have occurred in men having sex with men, and most patients have presented with proctitis.
Severe Tryptophan Starvation Blocks Onset of Conventional Persistence and Reduces Reactivation of Chlamydia trachomatis
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It is demonstrated that at low exogenous tryptophan concentrations a substantial fraction of C. trachomatis bacteria fail to traffic to the MTOC or to switch into the conventional persistent state in gamma interferon-induced human cells, which affects chlamydial development more rigorously than previously described.
Innate immunity in the male genital tract: Chlamydia trachomatis induces keratinocyte-derived chemokine production in prostate, seminal vesicle and epididymis/vas deferens primary cultures.
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This is the first report showing KC production by seminal vesicle cells after stimulation with TLR ligands, C. trachomatis or C. muridarum antigens, which indicates that different receptors of the innate immunity are present in the MGT.
Chlamydia pecorum Infection in the Male Reproductive System of Koalas (Phascolarctos cinereus)
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The study suggests that Chlamydia infection in the male reproductive tract is more widespread than originally thought and might be a reservoir for persistent chlamydial infections in koalas, with important implications for prophylactic strategies and epidemiology.
[Chlamydia infections of the male: what is relevant for urologists?].
TLDR
This review summarizes the most current developments in the diagnostics and therapeutical approaches in Chlamydia infections of the male accessory sex glands.
The Role of Chlamydia Trachomatis Infection in Young Men: What we Need to Know?
TLDR
There is a need for future studies and to diffuse the knowledge about Chlamydia trachomatis especially in urological clinical practice, in order to reduce the risk for failure diagnosis.
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    Der Hautarzt; Zeitschrift fur Dermatologie, Venerologie, und verwandte Gebiete
  • 2007
TLDR
Genital infections with Chlamydia trachomatis occur in all social groups in Germany and the occurrence of chlamydial antibodies in serum or in seminal fluid is not a sign of current infection.
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