Changes in human skeletal muscle contractility and hormone status during 2 weeks of heavy strength training

  title={Changes in human skeletal muscle contractility and hormone status during 2 weeks of heavy strength training},
  author={Truls Raastad and Trond Glomsheller and Trine Bj{\o}ro and Jostein Hall{\'e}n},
  journal={European Journal of Applied Physiology},
Abstract To examine neuromuscular and hormone changes during 2 weeks of heavy strength training, 18 weight-trained male students were recruited either into a heavy training group (HT, n=11) or into a control group (Ctr, n=7). The heavy training protocol consisted of leg-extensor workouts performed daily, while workouts were performed twice a week in the Ctr group. A test of one repetition maximum (1 RM) was performed before heavy training and on the 2nd day after heavy training. Isokinetic knee… 
Recovery of skeletal muscle contractility and hormonal responses to strength exercise after two weeks of high‐volume strength training
Two weeks of high volume strength training attenuated neuromuscular fatigue after a test workout with only minor changes in exercised induced hormone response.
Hypertrophy with unilateral resistance exercise occurs without increases in endogenous anabolic hormone concentration
In conclusion, unilateral training induced local muscle hypertrophy only in the exercised limb, which occurred in the absence of changes in systemic hormones that ostensibly play a role in musclehypertrophy.
Intense resistance training induces pronounced metabolic stress and impairs hypertrophic response in hind-limb muscles of rats
Rats submitted to 5 weeks of intensive resistance jump-training – high intensity, large volume, and short rest intervals – present high levels of blood corticosterone associated with negative effects on hypertrophy of types-I and II muscle fibers.
The effects of creatine supplementation on muscular performance and body composition responses to short-term resistance training overreaching
Some measures of muscular performance and body composition are enhanced to a greater extent following the rebound phase of short-term resistance training overreaching with creatine supplementation and these changes are not related to changes in circulating hormone concentrations obtained in the resting, postabsorptive state.
Effects of muscle contraction timing during resistance training on vascular function
Findings suggest that although both training does not deteriorate a vascular endothelial function, resistance training with quick lifting and slow lowering (that is, ERT) prevent the stiffening of arterial stiffness.
The effects of amino acid supplementation on hormonal responses to resistance training overreaching.
beta2-Adrenergic receptor downregulation and performance decrements during high-intensity resistance exercise overtraining.
Desensitization of the beta(2)-AR system suggests that this may be an important contributor to performance decrements due to excessive use of maximal resistance exercise loads.
Combined aerobic and resistance training and vascular function: effect of aerobic exercise before and after resistance training.
Results suggest that although vascular function is not improved by aerobic exercise before RT, performing aerobic exercise thereafter can prevent the deteriorating of vascular function.
The physiological and biomechanical bases of muscular hypertrophy/atrophy
Training with the MTC in a lengthened position is more effective for inducing enhanced training MTC adaptations, owing to internal mechanical and physiological stress in this position, and this loading method should be incorporated into a structured resistance training program for a range of populations.
High eccentric strength training reduces heart rate variability in healthy older men
The results of the present investigation suggest that high eccentric strength training performed by healthy older men increases peak torque and reduces systolic blood pressure, however, an autonomic imbalance towards sympathetic modulation predominance was induced by an unknown mechanism.