• Corpus ID: 83528142

Cellular polyploidy. Proliferation and differentiation.

@inproceedings{Brodskii1981CellularPP,
  title={Cellular polyploidy. Proliferation and differentiation.},
  author={V. Ya. Brodskii and I. V. Uryvaeva},
  year={1981}
}
21 Citations
ASSESSMENT OF INFLUENCE OF FOOD DYES ON DROSOPHILA MELANOGASTER MEIGH
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Synthetic food dyes Indigocarmine® and Ponso red® did not affect the life span of Drosophila melanogaster, but when introduced in high concentrations, they significantly reduced the fecundity of experimental insects.
Karyometric analysis of hepatocytes of common field mice from regions with different levels of technogenic contamination
Increased polyploidy of hepatocytes is shown using karyometric analysis in common field mice from different Altai regions with high levels of technogenic contamination of radiation and chemical
Role of estrogens in the regulation of prolactin receptors in liver cells of female rats
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Effect of Zosterin on Protein-Synthesizing Activity of Hepatocytes
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The drug modified the morphology and function of the nucleolar apparatus by changing the number and summary area of theucleoli, content of Ag proteins, nucleolar/nuclear ratio, and increased protein content in hepatocyte cytoplasm.
Tissue and Intracellular Reorganization of Rat Liver during Total Body Hypothermia
TLDR
Tissue and intracellular reorganization of the liver during total body hypothermia is studied and regeneration strategies at different levels of structural organization are evaluated.
Enzymic imprinting in adult animals
TLDR
It can be postulated that artificial enhancement of proliferation in the adult liver as a result of partial hepatectomy will modify the conditions that enable fetal and neonatal tissues to fix a level of enzyme activity modified through the action of inducers.
Effect of a low protein diet on restoration of the rat liver parenchyma after carbon tetrachloride poisoning
TLDR
The optimal protein content of the diet is one factor which determines the effectiveness of structural and functional compensation of the lost part of the cell population in the liver after injury.
Morphological manifestations of compensatory and adaptive processes in the liver during aging
TLDR
The results of morphometry and electron microscopy suggest that the compensatory-adaptive processes during hepatocyte ageing were mediated by intracellular regeneration, which led to cellular and nuclear hypertrophy similar to that observed in cells of static population (neurons, cardiomyocytes).
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