Cell autonomy of the mouse claw paw mutation.

  title={Cell autonomy of the mouse claw paw mutation.},
  author={Aysel Darbaş and Martine Jaegle and Erik T. Walbeehm and Hans van den Burg and Siska Driegen and Ludo A. M. Broos and Matthijs Uyl and Pim Visser and Frank Grosveld and Dies Meijer},
  journal={Developmental biology},
  volume={272 2},
Mice homozygous for the autosomal recessive mutation claw paw (clp) are characterized by limb posture abnormalities and congenital hypomyelination, with delayed onset of myelination of the peripheral nervous system but not the central nervous system. Although this combination of limb and peripheral nerve abnormalities in clp/clp mice might suggest a common neurogenic origin of the syndrome, it is not clear whether the clp gene acts primarily in the neurone, the Schwann cell or both. In the work… CONTINUE READING
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Axon–Schwann cell interactions during peripheral nerve degeneration and regeneration

  • S. S. Scherer, J. L. Salzer
  • Glial Cell Development. Bios Scientific…
  • 1996
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