Cardiac myocyte calcium transport in phospholamban knockout mouse: relaxation and endogenous CaMKII effects.

@article{Li1998CardiacMC,
  title={Cardiac myocyte calcium transport in phospholamban knockout mouse: relaxation and endogenous CaMKII effects.},
  author={Lin Li and Guoxiang Chu and Evangelia G Kranias and Donald M. Bers},
  journal={American journal of physiology. Heart and circulatory physiology},
  year={1998},
  volume={274 4},
  pages={H1335-H1347}
}
Increases in heart rate are accompanied by acceleration of relaxation. This effect is apparent at the single myocyte level and depends on sarcoplasmic reticulum (SR) Ca transport and Ca/calmodulin dependent protein kinase [CaMKII; see R. A. Bassani, A. Mattiazzi, and D. M. Bers. Am. J. Physiol. 268 ( Heart Circ. Physiol. 37): H703-H712, 1995]. Because phosphorylation of phospholamban (PLB) by CaMKII can stimulate SR Ca transport, it is a plausible candidate mechanism. We examined this issue… CONTINUE READING
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