Camptocormia (bent spine) in patients with Parkinson's disease—Characterization and possible pathogenesis of an unusual phenomenon

@article{Djaldetti1999CamptocormiaS,
  title={Camptocormia (bent spine) in patients with Parkinson's disease—Characterization and possible pathogenesis of an unusual phenomenon},
  author={Ruth Djaldetti and Ronit Mosberg-Galili and Haza Sroka and Doron Merims and Eldad Melamed},
  journal={Movement Disorders},
  year={1999},
  volume={14}
}
Camptocormia is characterized by severe forward flexion of the thoracolumbar spine which increases while walking and disappears in the recumbent position. We describe for the first time eight patients with presumed idiopathic Parkinson's disease (mean age 66 ± 5 yrs; mean symptom duration 13.1 ± 5.1 yrs) who developed camptocormia. This impressive abnormal posture emerged 4–14 years from disease onset, and in some patients stooped posture was the prominent symptom at diagnosis. There was no… Expand
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TLDR
The following case, illustrating the marked improvement of severe thoracolumbar spine flexion after pallidotomy, may support the hypothesis of dystonic origin of camptocormia in PD. Expand
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TLDR
There is no established consensus for treatment of camptocormia in Parkinson's disease, but there are non-pharmacological, pharmacological and surgical approaches that can be used. Expand
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TLDR
There are 3 main hypotheses on the pathogenesis of CC in PD, i.e. a focal myopathy of the trunk muscles, axial dystonia and a drug-induced etiology, which are given a synopsis and critical acclaim. Expand
Camptocormia in a patient with Parkinson disease and a myopathy with nemaline rods.
TLDR
A 77-yr-old male patient who was followed up with the diagnosis of rheumatoid arthritis for 2 yrs was admitted with progressive gait deterioration and hyperflexion of trunk, disappearing in supine position, was detected and diagnosed as camptocormia. Expand
Camptocormia in Parkinson’s disease
TLDR
In a few patients, the abnormal posture improved and in others it was unaltered or even became worse following levodopa administration, but in most, the severity of camptocormia was unchanged during the “on” and “off” phases. Expand
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TLDR
A patient is reported who had a marked camptocormia prior to the onset of clinical triad of Parkinsonism and seems to be related to the clinical severity of PD. Expand
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TLDR
Etiologic classification of camptocormia is proposed and it is concluded that this heterogeneous disorder has multiple etiologies and variable response to systemic and local therapies. Expand
Camptocormia in Parkinson's disease mimicked by focal myositis of the paraspinal muscles
TLDR
The case of a 63‐year‐old man with idiopathic Parkinson's disease who developed kyphosis and a severe forward flexion of the thoracolumbar spine indicates that camptocormia can be mimicked by focal myositis of paraspinal muscles and must be included in the differential diagnosis, especially when additional symptoms as inflammatory signs or weakness are present. Expand
[Camptocormia in patients with Parkinson's disease].
TLDR
Camptocormia leading to malposition, increasing postural instability and risk of falling in Parkinson's disease patients is characterized by an abnormal posture with involuntary forward flexion of the trunk which appears in erect position, increases during prolonged standing or walking, and abates in supine position. Expand
Parkinson’s disease with camptocormia
TLDR
Patients with camptocormia were characterised by prominent levodopa-unresponsive axial symptoms (ie, axial rigidity, gait disorder and postural instability), along with a tendency for greater error in the antisaccade paradigm, and it is suggested that the salient features of parkinsonism observed in patients with camps are likely to represent a specific form of Parkinson’s disease. Expand
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