Calcium responses to caffeine and muscarinic receptor agonists are altered in traumatically injured neurons.

@article{Weber2002CalciumRT,
  title={Calcium responses to caffeine and muscarinic receptor agonists are altered in traumatically injured neurons.},
  author={John T. Weber and Beverly A. Rzigalinski and Earl F. Ellis},
  journal={Journal of neurotrauma},
  year={2002},
  volume={19 11},
  pages={1433-43}
}
A fundamental mechanism that is believed to contribute to neuronal injury and death following traumatic brain injury (TBI) is a disruption in cellular calcium homeostasis. Of primary importance to these homeostatic mechanisms are intracellular calcium stores located on the endoplasmic reticulum. These intracellular stores play an important role in maintaining normal levels of calcium and calcium-mediated signaling through these stores is critical to several physiological processes in neurons… CONTINUE READING
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