Calbindin D-28k and parvalbumin immunoreactivity in the frontal cortex in patients with frontal lobe dementia of non-Alzheimer type associated with amyotrophic lateral sclerosis.

@article{Ferrer1993CalbindinDA,
  title={Calbindin D-28k and parvalbumin immunoreactivity in the frontal cortex in patients with frontal lobe dementia of non-Alzheimer type associated with amyotrophic lateral sclerosis.},
  author={Isisdre Ferrer and Teresa Tu{\~n}{\'o}n and Mar{\'i}a Trinidad Serrano and Rachel Casas and Salvador Alc{\'a}ntara and M. J. Z{\'u}jar and R M Rivera},
  journal={Journal of neurology, neurosurgery, and psychiatry},
  year={1993},
  volume={56 3},
  pages={257-61}
}
The morphology and distribution of local-circuit neurons (interneurons) were examined, by calbindin D-28k and parvalbumin immunocytochemistry, in the frontal cortex (area 8) in two patients with frontal lobe dementia of non-Alzheimer type associated with classical amyotrophic lateral sclerosis (ALS), and in seven normal cases. The density of calbindin D-28k immunoreactive cells was dramatically reduced in ALS patients, but the density of parvalbumin-immunoreactive neurons was preserved… CONTINUE READING
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