Caffeine-Induced Psychosis

  title={Caffeine-Induced Psychosis},
  author={Dawson W. Hedges and Fu Lye M Woon and Scott P Hoopes},
  journal={CNS Spectrums},
  pages={127 - 131}
ABSTRACT As a competitive adenosine antagonist, caffeine affects dopamine transmission and has been reported to worsen psychosis in people with schizophrenia and to cause psychosis in otherwise healthy people. We report of case of apparent chronic caffeine-induced psychosis characterized by delusions and paranoia in a 47-year-old man with high caffeine intake. The psychosis resolved within 7 weeks after lowering caffeine intake without use of antipsychotic medication. Clinicians might consider… 
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It is suggested that caffeine consumption among patients with mood or psychotic symptoms should be assessed carefully in clinical practice as part of routine psychiatric evaluations.
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The patient represented after two months with similar acute psychotic symptoms in the background of increasing coffee consumption, and was discharged to his SRF with recommendations to monitor and manage his coffee intake.
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A previously healthy 29-year-old man who developed an abrupt onset of paranoid delusions, thought disorganization, and bizarre behavior after increasing his caffeine intake to >1500 mg per day over a period of 2 days supports the existing data on psychosis precipitated by excessive amounts of caffeine intake.
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Advice on the treatment of acute and prolonged psychotic illness in relationship to drug use, including delirium tremens caused by withdrawal from alcohol or sedatives is given.
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    Journal of Alzheimer's disease : JAD
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Caffeine can be regarded as a pharmacological tool to increase energy and effortful behavior in daily activities and its putative therapeutic effects on depression and ADHD have been insufficiently studied.
Caffeine Is A Stimulant But It May Not Be A Psychostimulant
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Psychopathology Related to Energy Drinks: A Psychosis Case Report
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  • Psychology, Medicine
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The uses and effects of caffeine as a psychoactive drug in chronic psychiatric inpatient groups are described, and deleterious effects may result from the interaction of caffeine with commonly prescribed psychotropic drugs.
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This review article aims to address the specific research studies and case reports that relate caffeine to psychiatric symptoms by recognizing, educating, and treating patients using a tapering approach.
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Smoking and excessive caffeine use are associated with poor therapeutic responses to clozapine and should be considered in the pharmacological management of treatment-refractory psychosis, regardless of the primary diagnosis.
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The present review focuses on the effects of caffeine on striatal signal transduction and on their involvement in caffeine-mediated motor stimulation.
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Issues related to the epidemiology, nosology, and differential diagnosis of organic aggressive disorders in the elderly are discussed and an approach for using beta-blockers, anticonvulsant agents, and serotonin-specific antidepressants and an antianxiety agent for the treatment of chronic aggression is presented.
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It is concluded that for the healthy adult population, moderate daily caffeine intake is not associated with adverse effects such as general toxicity, cardiovascular effects, effects on bone status and calcium balance, changes in adult behaviour, increased incidence of cancer and effects on male fertility.