Brief secondhand smoke exposure depresses endothelial progenitor cells activity and endothelial function: sustained vascular injury and blunted nitric oxide production.

@article{Heiss2008BriefSS,
  title={Brief secondhand smoke exposure depresses endothelial progenitor cells activity and endothelial function: sustained vascular injury and blunted nitric oxide production.},
  author={Christian Heiss and Nicolas Amabile and Andrew C. Y. Lee and Wendy May Real and Suzaynn Francine Schick and David Lao and Maelene L Wong and Sarah Jahn and Franca S. Angeli and Petros Minasi and Matthew L. Springer and Sally Katharine Hammond and Stanton A. Glantz and William F Grossman and John R Balmes and Yerem Yeghiazarians},
  journal={Journal of the American College of Cardiology},
  year={2008},
  volume={51 18},
  pages={1760-71}
}
OBJECTIVES This study sought to analyze the effects of acute secondhand smoke (SHS) exposure on the number and function of endothelial progenitor cells (EPCs) over 24 h. BACKGROUND Secondhand smoke increases the risk of vascular disease and is a major public health concern, but the mechanism(s) of action are not fully understood. METHODS Healthy nonsmokers (age SEM 30.3 +/- 1.3 years, n = 10) were exposed to 30 min of SHS yielding cotinine levels commonly observed in passive smokers and to… CONTINUE READING
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