Bone resorption caused by three periodontal pathogens in vivo in mice is mediated in part by prostaglandin.

@article{Zubery1998BoneRC,
  title={Bone resorption caused by three periodontal pathogens in vivo in mice is mediated in part by prostaglandin.},
  author={Y Zubery and Colin R. Dunstan and B M Story and Lakshmyya N. Kesavalu and Jeffrey L. Ebersole and Stanley C. Holt and Brendan F. Boyce},
  journal={Infection and immunity},
  year={1998},
  volume={66 9},
  pages={
          4158-62
        }
}
Gingival inflammation, bacterial infection, alveolar bone destruction, and subsequent tooth loss are characteristic features of periodontal disease, but the precise mechanisms of bone loss are poorly understood. Most animal models of the disease require injury to gingival tissues or teeth, and the effects of microorganisms are thus complicated by host responses to tissue destruction. To determine whether three putative periodontal pathogens, Porphyromonas gingivalis, Campylobacter rectus, and… CONTINUE READING

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