Biophysical regulation of Chlamydia pneumoniae-infected monocyte recruitment to atherosclerotic foci

@inproceedings{Evani2016BiophysicalRO,
  title={Biophysical regulation of Chlamydia pneumoniae-infected monocyte recruitment to atherosclerotic foci},
  author={Shankar Jaikishan Evani and Anand Kumar Ramasubramanian},
  booktitle={Scientific reports},
  year={2016}
}
Chlamydia pneumoniae infection is implicated in atherosclerosis although the contributory mechanisms are poorly understood. We hypothesize that C. pneumoniae infection favors the recruitment of monocytes to atherosclerotic foci by altering monocyte biophysics. Primary, fresh human monocytes were infected with C. pneumoniae for 8 h, and the interactions between monocytes and E-selectin or aortic endothelium under flow were characterized by video microscopy and image analysis. The distribution of… CONTINUE READING

References

Publications referenced by this paper.
Showing 1-10 of 51 references

the hypothesis is still untested

  • Grayston, J. T. et al. Infection with Chlamydia pneumoniae as a disease
  • Pathog Dis. 73, 1–9
  • 2015

A clinical issue

  • R. Sessa, M. Di Pietro, S. FIlardo, Turriziani, O. Infectious burden, atherosclerosis
  • World J Clin Cases 2, 240–249
  • 2014

Similar Papers

Loading similar papers…