Bicarbonate-rich choleresis induced by secretin in normal rat is taurocholate-dependent and involves AE2 anion exchanger.

@article{Banales2006BicarbonaterichCI,
  title={Bicarbonate-rich choleresis induced by secretin in normal rat is taurocholate-dependent and involves AE2 anion exchanger.},
  author={Jes{\'u}s Mar{\'i}a Banales and Fabi{\'a}n Arenas and Carlos M. Rodr{\'i}guez-Ortigosa and Elena Dur{\'e}ndez S{\'a}ez and Iker Uriarte and Richard Doctor and Jes{\'u}s Prieto and Juan Francisco Medina},
  journal={Hepatology},
  year={2006},
  volume={43 2},
  pages={266-75}
}
Canalicular bile is modified along bile ducts through reabsorptive and secretory processes regulated by nerves, bile salts, and hormones such as secretin. Secretin stimulates ductular cystic fibrosis transmembrane conductance regulator (CFTR)-dependent Cl- efflux and subsequent biliary HCO3- secretion, possibly via Cl-/HCO3- anion exchange (AE). However, the contribution of secretin to bile regulation in the normal rat, the significance of choleretic bile salts in secretin effects, and the role… CONTINUE READING

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