BAS1: A gene regulating brassinosteroid levels and light responsiveness in Arabidopsis.

@article{Neff1999BAS1AG,
  title={BAS1: A gene regulating brassinosteroid levels and light responsiveness in Arabidopsis.},
  author={Michael M. Neff and Steve M Nguyen and Elizabeth J. Malancharuvil and Shozo Fujioka and Takahiro Noguchi and Hideharu Seto and Masayoshi Tsubuki and Takeshi Honda and Suguru Takatsuto and Satoko Yoshida and Joanne Chory},
  journal={Proceedings of the National Academy of Sciences of the United States of America},
  year={1999},
  volume={96 26},
  pages={15316-23}
}
The Arabidopsis bas1-D mutation suppresses the long hypocotyl phenotype caused by mutations in the photoreceptor phytochrome B (phyB). The adult phenotype of bas1-D phyB-4 double mutants mimics that of brassinosteroid biosynthetic and response mutants. bas1-D phyB-4 has reduced levels of brassinosteroids and accumulates 26-hydroxybrassinolide in feeding experiments. The basis for the mutant phenotype is the enhanced expression of a cytochrome P450 (CYP72B1). bas1-D suppresses a phyB-null allele… CONTINUE READING
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Development (Cambridge, U.K

  • Q. Tian, J. W. Reed
  • 1999

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