B antigen acquired by normal A1 red cells exposed to a patient's serum

@article{Stayboldt1987BAA,
  title={B antigen acquired by normal A1 red cells exposed to a patient's serum},
  author={Carla Stayboldt and Ann Rearden and Thomas A. Lane},
  journal={Transfusion},
  year={1987},
  volume={27}
}
The authors report a patient with the acquired B antigen whose serum contained a factor capable of inducing the acquired B phenotype on normal A1 red cells in vitro. The patient's red cells were not, however, agglutinated by a monoclonal anti‐B. These findings and the abolition of the acquired B antigen by treatment of the patient's red cells with acetic anhydride support the deacetylation theory of the genesis of the acquired B antigen. 
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TLDR
By agglutination kinetics using an immune anti‐B, the acquired B reactive structure was found to differ from that of a normal B, points out to the part played by one electrically charged chemical group.
Atypical Polyagglutination Associated with an Acquired B Antigen
The finding of an acquired B antigen, together with polyagglutination, in an elderly man led to studies indicating adsorption of bacterial material as the likely cause of both of these red blood cell
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TLDR
Evidence is presented for the existence of an additional type of weak B antigen developing de novo.
A Weak B Antigen, Probably Acquired
TLDR
W"at'vPva -rov His cells were not agglutinrated, and the reactions varied from + to + + + in strength and were inhibited by gronp. ",Case 29.
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The biochemical influences at work in wound healing are quoted to indicate the complicated nature of such problems, and it is suggested that if research is to be undertaken along these lines in a clinical unit it is no good entrusting it to a clinician who dabbles in biochemistry.
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Abstract. The chemical acetylation of RBC bearing the acquired B antigen led to the disappearance of the agglutinability by anti‐B and restored the A1 specificity. The same results are obtained using
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TLDR
Results of tests using purified Arachis hypogaea and Bandeiraea simplicifolia lectins, and red blood cells either T, Tk or both T and Tk‐activated by bacterial enzymes in vitro, suggest that the acquired‐B phenomenon, T‐activation and T k‐activation result from the action of different bacterial enzymes.
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TLDR
Red cell sensitization with B active E. coli O86 substances was achieved in vivo in a minority of severely diseased infants and in germ-free and ordinary chicks which were in tourniquet shock after treatment with cathartics.
ENZYMES OF CLOSTRIDIUM TERTIUM
The enzymatic degradation of purified blood group substances represents an increasingly fruitful approach to the elucidation of their immunochemical specificity, and progress to date has recently
Effect of Modification of Amino Groups of Human Erythrocytes on M, N and NVg Blood Group Specificities
TLDR
The results suggest that the difference between M and N receptors is not determined by the structure of oligosaccharide chains and only slightly decreased agglutinability by human anti‐H, anti‐A and anti‐I sera.
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