Argyrophil cell hyperplasia of fundic mucosa in patients with chronic atrophic gastritis.

@article{Bordi1986ArgyrophilCH,
  title={Argyrophil cell hyperplasia of fundic mucosa in patients with chronic atrophic gastritis.},
  author={Cesare Bordi and Francesco Paolo Pilato and Giuseppe Carfagna and C. Ferrari and Tiziana D’adda and Roberto Sivelli and A. M. Bertel{\`e} and G. Missale},
  journal={Digestion},
  year={1986},
  volume={35 Suppl 1},
  pages={
          130-43
        }
}
Eighteen cases of severe hyperplasia of fundic argyrophil cells observed during routine histologic examination of endoscopic gastric biopsy specimens from unselected patients with upper gastro-intestinal symptomatology were investigated. All patients, except one, were female with a mean age of 57 years. Atrophic gastritis of fundic mucosa with severe hypo- or achlorhydria was present in all cases. Hypergastrinaemia (of antral origin) was found in 15 subjects in which circulating gastrin levels… 
Ultrastructural characterization of fundic endocrine cell hyperplasia associated with atrophic gastritis and hypergastrinaemia
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Fundic endocrine cell hyperplasia occurring inhypergastrinaemic CAG is in most cases cytologically similar to that found in other hypergastrinemic conditions, in which the gastrin-dependent ECL cells were already found to prevail, which explains why fundic carcinoids arising in CAG are mostly composed of E CL cells.
Argyrophil cell hyperplasia associated with chronic corpus gastritis in gastric ulcer disease.
TLDR
It is concluded that focal AC hyperplasia is a common phenomenon in gastric ulcer patients and is inherently related to the spontaneous development of atrophy in the corpus mucosa of these patients.
Endocrine cell growths in atrophic body gastritis. Critical evaluation of a histological classification
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Evaluation of the correspondence of the classification of non‐antral endocrine cell growths proposed by Solcia and co‐workers with clinical features and non‐endocrine mucosal changes shows clinical relevance without the need to perform expensive, time‐consuming morphometric evaluations.
Chromogranin A – containing endocrine cells in the gastric mucosa of patietns with Helicobacter pylori associated chronic gastritis type B -
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A close correlation between an decrease in gastric chromogranin levels and the histologic activity was also present, suggesting that certain peptide-immune reactions in the gastric mucosa exist in HP infection.
Pattern of gastric endocrine cells in microcarcinoidosis — an immunohistochemical study of 14 gastric biopsies
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In conclusion, invasive ECL carcinoids seem to be clinically less dangerous than was thought until recently and microcarcinoid gastric biopsies about 0.5 cm in size, are suggestive of adjacent invasive carcinoid.
Carcinoid (ECL Cell) Tumor of the Oxyntic Mucosa of the Stomach: A Hormone-Dependent Neoplasm?
TLDR
It is proposed that hypergastrinemia is responsible for the hyperplastic response of endocrine cells of the oxyntic mucosa in both chronic atrophic gastritis and ZES, and the subsequent study of single cases of fundic carcinoid tumors extended this hypothesis by linkinghypergastRinemia to the development of neoplasms from the same endocrine cell population.
Quantitative electron microscopy of endocrine cells in oxyntic mucosa of normal human stomach
TLDR
The quantitative ultrastructural analysis of the endocrine cell population of the normal human oxyntic mucosa of the stomach in gastric biopsies collected from five male and five female healthy volunteers aged 19–31 may allow a better evaluation of physiological and pharmacological variations of the ENDO cell population.
Hyperplastic proliferations of the ECL cells.
  • Y. Dayal
  • Medicine
    The Yale journal of biology and medicine
  • 1992
TLDR
Such carcinoids arising in patients with chronic hypergastrinemia differ both in their clinical and pathologic profiles from the sporadic carcinoids that occur in normogastrimenic individuals and, therefore, need to be distinguished from them.
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