Annotation of long non-coding RNAs expressed in collaborative cross founder mice in response to respiratory virus infection reveals a new class of interferon-stimulated transcripts.

@article{Josset2014AnnotationOL,
  title={Annotation of long non-coding RNAs expressed in collaborative cross founder mice in response to respiratory virus infection reveals a new class of interferon-stimulated transcripts.},
  author={Laurence Josset and Nicolas Tchitchek and Lisa E. Gralinski and Martin T. Ferris and Amie J. Eisfeld and Richard Green and Matthew J. Thomas and Jennifer Tisoncik-Go and Gary P. Schroth and Yoshihiro Kawaoka and Fernando Pardo Manuel de Villena and Ralph S. Baric and Mark T. Heise and Xinxia Peng and Michael G. Katze},
  journal={RNA biology},
  year={2014},
  volume={11 7},
  pages={875-90}
}
The outcome of respiratory virus infection is determined by a complex interplay of viral and host factors. Some potentially important host factors for the antiviral response, whose functions remain largely unexplored, are long non-coding RNAs (lncRNAs). Here we systematically inferred the regulatory functions of host lncRNAs in response to influenza A virus and severe acute respiratory syndrome coronavirus (SARS-CoV) based on their similarity in expression with genes of known function. We… CONTINUE READING

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Here we systematically inferred the regulatory functions of host lncRNAs in response to influenza A virus and severe acute respiratory syndrome coronavirus ( SARS - CoV ) based on their similarity in expression with genes of known function .
Here we systematically inferred the regulatory functions of host lncRNAs in response to influenza A virus and severe acute respiratory syndrome coronavirus ( SARS - CoV ) based on their similarity in expression with genes of known function .
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