Angiotensin II inhibition during myocardial ischemia-reperfusion in dogs: effects on leukocyte infiltration, nitric oxide synthase isoenzymes activity and left ventricular ejection fraction.

@article{Gusmo2005AngiotensinII,
  title={Angiotensin II inhibition during myocardial ischemia-reperfusion in dogs: effects on leukocyte infiltration, nitric oxide synthase isoenzymes activity and left ventricular ejection fraction.},
  author={Frederick Malta Buarque de Gusm{\~a}o and Cl{\'a}udia Becker and Maria Helena Catelli de Carvalho and Luiz Fernando Monteiro Barros},
  journal={International journal of cardiology},
  year={2005},
  volume={100 3},
  pages={363-70}
}
Leukocyte infiltration and activation in myocardial reperfusion injury may be modulated by nitric oxide synthase isoforms. Angiotensin II influences leukocyte activation directly or by nitric oxide generation mechanisms. The effects of angiotensin II inhibition before reperfusion on myocardial function, leukocyte accumulation and nitric oxide synthase were evaluated on three groups of eight dogs. They were submitted to occlusion of the left anterior descending coronary artery for 90 min… CONTINUE READING

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