Angiotensin II Infusion–Induced Inflammation, Monocytic Fibroblast Precursor Infiltration, and Cardiac Fibrosis are Pressure Dependent

Abstract

The activated renin–angiotensin–aldosterone system increases blood pressure and intracellular signals, thus leading to cardiac fibrosis. Whether increased blood pressure or angiotensin II-activated signaling is responsible for elevated angiotensin II–induced cardiac remodeling is unknown. Here, we aimed to determine whether lowering blood pressure with… (More)
DOI: 10.1007/s12012-011-9109-z

Topics

6 Figures and Tables

Slides referencing similar topics