An overview on peripheral vascular disease in blackfoot disease-hyperendemic villages in Taiwan.

@article{Tseng2002AnOO,
  title={An overview on peripheral vascular disease in blackfoot disease-hyperendemic villages in Taiwan.},
  author={Chin-Hsiao Tseng},
  journal={Angiology},
  year={2002},
  volume={53 5},
  pages={529-37}
}
The arsenic-related peripheral vascular disease found to be endemic along the southwestern coast of Taiwan is reviewed. In the early 20th century a strange disease involving the lower extremities characterized by typical clinical symptoms and signs of progressive arterial occlusion was reported in a confined area located along the southwestern coast of Taiwan. The disease was locally called "blackfoot disease" because of its gangrenous appearance involving the feet of the patients. The… CONTINUE READING

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However , its interaction with some trace elements and its association with hypertension and diabetes mellitus could also explain part of its higher risk of developing atherosclerosis .
However , its interaction with some trace elements and its association with hypertension and diabetes mellitus could also explain part of its higher risk of developing atherosclerosis .
However , its interaction with some trace elements and its association with hypertension and diabetes mellitus could also explain part of its higher risk of developing atherosclerosis .
However , its interaction with some trace elements and its association with hypertension and diabetes mellitus could also explain part of its higher risk of developing atherosclerosis .
The atherogenicity of arsenic could be associated with its effects on hypercoagulability , endothelial injury , smooth muscle cell proliferation , somatic mutation , oxidative stress , and apoptosis .
The atherogenicity of arsenic could be associated with its effects on hypercoagulability , endothelial injury , smooth muscle cell proliferation , somatic mutation , oxidative stress , and apoptosis .
The atherogenicity of arsenic could be associated with its effects on hypercoagulability , endothelial injury , smooth muscle cell proliferation , somatic mutation , oxidative stress , and apoptosis .
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