An enzymatic ruler modulates Lewis antigen glycosylation of Helicobacter pylori LPS during persistent infection.

@article{Nilsson2006AnER,
  title={An enzymatic ruler modulates Lewis antigen glycosylation of Helicobacter pylori LPS during persistent infection.},
  author={C. Nilsson and Anna Skoglund and A. Moran and H. Annuk and L. Engstrand and S. Normark},
  journal={Proceedings of the National Academy of Sciences of the United States of America},
  year={2006},
  volume={103 8},
  pages={
          2863-8
        }
}
Helicobacter pylori persistently colonizes about half the human population and contributes to the development of peptic ulcer disease and gastric cancer. This organism has evolved means to structurally alter its surface characteristics to evade innate and adaptive immune responses. H. pylori produces LPS O-antigen units that can be posttranslationally fucosylated to generate Lewis antigens, structures also found on human epithelial cells. We demonstrate an extensive diversity of Lewis x and… Expand
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