Amyloid-β oligomers unveil a novel primate model of sporadic Alzheimer's disease

Abstract

The development and characterization of Alzheimer’s disease (AD) animal models that faithfully recapitulate key components of the pathogenic process has been a major research focus for nearly a quarter of a century. Neuropathologically, AD is characterized by the accumulation of extracellular amyloid-β (Aβ) plaques, intracellular neurofibrillary tangles… (More)
DOI: 10.3389/fnins.2015.00047

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Cite this paper

@inproceedings{Jebelli2015AmyloidOU, title={Amyloid-β oligomers unveil a novel primate model of sporadic Alzheimer's disease}, author={Joseph D. Jebelli and Thomas Piers}, booktitle={Front. Neurosci.}, year={2015} }