Altered factor VII activating protease expression in murine hepatic fibrosis and its influence on hepatic stellate cells.

Factor VII-activating protease (FSAP): vascular functions and role in atherosclerosis.

Expression of MMPs and TIMPs in liver fibrosis - a systematic review with special emphasis on anti-fibrotic strategies.

Local inhibition of liver fibrosis by specific delivery of a platelet-derived growth factor kinase inhibitor to hepatic stellate cells.

Mechanisms of fibrinolysis in chronic liver injury (with special emphasis on MMPs and TIMPs).

Models of liver fibrosis: exploring the dynamic nature of inflammation and repair in a solid organ.

Nucleic acids potentiate Factor VII-activating protease (FSAP)-mediated cleavage of platelet-derived growth factor-BB and inhibition of vascular smooth muscle cell proliferation.

Pro-fibrogenic potential of PDGF-D in liver fibrosis.

The myofibroblast: one function, multiple origins.

A positively charged cluster in the epidermal growth factor-like domain of Factor VII-activating protease (FSAP) is essential for polyanion binding.

Changes of the hepatic proteome in murine models for toxically induced fibrogenesis and sclerosing cholangitis.

Altered factor VII activating protease expression in murine hepatic fibrosis and its influence on hepatic stellate cells.

Hydrolysis of Hyaluronic Acid in Lymphedematous Tissue Alleviates Fibrogenesis via TH1 Cell-Mediated Cytokine Expression

Factor VII activating protease (FSAP): a novel protective factor in liver fibrosis.

Factor VII activating protease (FSAP) exerts anti-inflammatory and anti-fibrotic effects in liver fibrosis in mice and men.

High negative charge-to-size ratio in polyphosphates and heparin regulates factor VII-activating protease.

The Marburg I variant (G534E) of the factor VII-activating protease determines liver fibrosis in hepatitis C infection by reduced proteolysis of platelet-derived growth factor BB.

Factor VII-activating protease (FSAP): vascular functions and role in atherosclerosis.

Expression of MMPs and TIMPs in liver fibrosis - a systematic review with special emphasis on anti-fibrotic strategies.

Local inhibition of liver fibrosis by specific delivery of a platelet-derived growth factor kinase inhibitor to hepatic stellate cells.

Mechanisms of fibrinolysis in chronic liver injury (with special emphasis on MMPs and TIMPs).

Models of liver fibrosis: exploring the dynamic nature of inflammation and repair in a solid organ.

Nucleic acids potentiate Factor VII-activating protease (FSAP)-mediated cleavage of platelet-derived growth factor-BB and inhibition of vascular smooth muscle cell proliferation.

Pro-fibrogenic potential of PDGF-D in liver fibrosis.

The myofibroblast: one function, multiple origins.

A positively charged cluster in the epidermal growth factor-like domain of Factor VII-activating protease (FSAP) is essential for polyanion binding.

Changes of the hepatic proteome in murine models for toxically induced fibrogenesis and sclerosing cholangitis.

Altered factor VII activating protease expression in murine hepatic fibrosis and its influence on hepatic stellate cells.

Hydrolysis of Hyaluronic Acid in Lymphedematous Tissue Alleviates Fibrogenesis via TH1 Cell-Mediated Cytokine Expression

Factor VII activating protease (FSAP): a novel protective factor in liver fibrosis.

Factor VII activating protease (FSAP) exerts anti-inflammatory and anti-fibrotic effects in liver fibrosis in mice and men.

High negative charge-to-size ratio in polyphosphates and heparin regulates factor VII-activating protease.

The Marburg I variant (G534E) of the factor VII-activating protease determines liver fibrosis in hepatitis C infection by reduced proteolysis of platelet-derived growth factor BB.

Factor VII-activating protease (FSAP): vascular functions and role in atherosclerosis.

Expression of MMPs and TIMPs in liver fibrosis - a systematic review with special emphasis on anti-fibrotic strategies.

Local inhibition of liver fibrosis by specific delivery of a platelet-derived growth factor kinase inhibitor to hepatic stellate cells.

Mechanisms of fibrinolysis in chronic liver injury (with special emphasis on MMPs and TIMPs).

Models of liver fibrosis: exploring the dynamic nature of inflammation and repair in a solid organ.

Nucleic acids potentiate Factor VII-activating protease (FSAP)-mediated cleavage of platelet-derived growth factor-BB and inhibition of vascular smooth muscle cell proliferation.

Pro-fibrogenic potential of PDGF-D in liver fibrosis.

The myofibroblast: one function, multiple origins.

A positively charged cluster in the epidermal growth factor-like domain of Factor VII-activating protease (FSAP) is essential for polyanion binding.

Changes of the hepatic proteome in murine models for toxically induced fibrogenesis and sclerosing cholangitis.