Alcohol-induced depersonalization

@article{Raimo1999AlcoholinducedD,
  title={Alcohol-induced depersonalization},
  author={E B Raimo and Richard A. Roemer and Mark L. Moster and Yang Shan},
  journal={Biological Psychiatry},
  year={1999},
  volume={45},
  pages={1523-1526}
}
Medication-associated Depersonalization Symptoms: Report of Transient Depersonalization Symptoms Induced by Minocycline
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Medication-associated depersonalization symptoms typically resolve once the inducing drug has been withdrawn, and the postulated pathogenesis by which some of these drugs induced depersonALization symptoms is discussed.
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Depersonalization appears to be associated with functional abnormalities along sequential hierarchical areas, secondary and cross-modal, of the sensory cortex (visual, auditory, and somatosensory), as well as areas responsible for an integrated body schema.
[Depersonalisation/derealization - clinical picture, diagnostics and therapy].
  • M. Michal, M. Beutel
  • Medicine
    Zeitschrift fur Psychosomatische Medizin und Psychotherapie
  • 2009
TLDR
There is a considerable lack of empirical research onDP-DR with respect to the health-care situation of depersonalized patients and with regard to treatment options, and it is suggested that symptoms of DP-DR indicate disease severity and negatively predict therapy outcome.
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Patients with peripheral Vestibular disease have a deficit in the ability to update orientation on the environment and a high prevalence of DD symptoms, which may imply a high order effect of the vestibular impairment.
Dissociation, Resting EEG, and Subjective Sleep Experiences in Undergraduates
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It is proposed that suppression in the α band and raised levels of &thgr; activity, which are typical for high dissociators, might help to explain why dissociative symptoms are accompanied by attentional and memory deficits.
Striking Similarities between Clinical and Biological Properties of Ketamine and Ethanol: Linking Antidepressant-After Effect and Burgeoning Addiction?
TLDR
These overlaps are mainly addressed here in more detail, what may draw the reader in terms of the treatment of mood disorders to both, the possibility of a progressing transfer from ADE to addiction when repeatedly using therapeutic ketamine pulses, and a hypothesized therapeutic `antidepressant window´ of modest and cautious ethanol use in depressives, who are (still?) not addicted to ethanol.
Adaptación y validación al castellano de la Escala de Despersonalización de Cambridge
INTRODUCCION: La Escala de Despersonalizacion de Cambridge es un cuestionario autoadministrativo cuyo objetivo es evaluar la frecuencia y duracion de los sintomas de despersonalizacion en los ultimos
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References

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TLDR
These cases suggest that after the patients had experienced depersonalization, external stressors and intrapsychic factors may have contributed to its continued use as a defense mechanism after marijuana use.
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It is concluded that the depersonalization syndrome is a distinct entity of which these cases are examples, and the differential diagnosis and treatment are discussed.
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The data indicate that depersonalisation results from anxiety; it follows an anxiety attack and is successfully treated with anxiolytic drugs, and in the case of endogenous depression, depersonalisaticm leads to lingering depressive phase, increasing the patients resistance to antidepressive therapy.
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The authors report on the results of a preliminary investigation of the quantitative electroencephalographic (QEEG) correlates of severe DSM-III-R crack cocaine dependence in seven patients abstinent from cocaine for 1 to 68 days, finding increased absolute and relative alpha power.
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TLDR
The successful treatment of a case with desipramine is reported and it is suggested that, because there is a link between depersonalization and anxiety disorders, tricyclic antidepressants may prove effective for depERSONalization.
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