Airway mucosal thickening and bronchial hyperresponsiveness induced by inhaled beta 2-agonist in mice.

@article{Tamaoki2004AirwayMT,
  title={Airway mucosal thickening and bronchial hyperresponsiveness induced by inhaled beta 2-agonist in mice.},
  author={Jun Tamaoki and Etsuko Tagaya and Kiyomi Kawatani and Junko Nakata and Yumie Endo and Atsushi Nagai},
  journal={Chest},
  year={2004},
  volume={126 1},
  pages={
          205-12
        }
}
BACKGROUND Patients with chronic persistent asthma require frequent use of inhaled beta(2)-agonist, which may result in aggravation of asthma symptoms. Our recent in vitro study has shown that beta(2)-agonist stimulates the growth of human airway epithelial cell lines. STUDY OBJECTIVE To determine whether beta(2)-agonist likewise affects airway epithelial cell proliferation in vivo and, if so, what the mechanism of action is, we examined the effect of salbutamol on the morphology of murine… CONTINUE READING
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