Ageing of Neurospora crassa. VI. Cytochemical and cytological correlates of senescence in three model systems

@article{Rana1978AgeingON,
  title={Ageing of Neurospora crassa. VI. Cytochemical and cytological correlates of senescence in three model systems},
  author={Rajendra S. Rana and Kenneth D. Munkres},
  journal={Mechanisms of Ageing and Development},
  year={1978},
  volume={7},
  pages={273-288}
}
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9 Citations
Background Citations
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Methods Citations
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9 Citations

Ageing of Neurospora crassa. V. Lipid peroxidation and decay of respiratory enzymes in an inositol auxotroph

Ageing of Neurospora crassa. VII. Accumulation of fluorescent pigment (Lipofuscin) and inhibition of the accumulation by nordihydroguaiaretic acid

Antioxidants prolong life span and inhibit the senescence- dependent accumulation of fluorescent pigment (lipofuscin) in clones of Podospora anserina

Inositol-Limited Growth, Repair, and Translocation in an Inositol-Requiring Mutant of Neurospora crassa

These results are consistent with cell lysis occurring in the mutant grown on limiting inositol because of a structurally weakened cell wall and membrane deterioration.

Unstable mitochondrial DNA in natural-death nuclear mutants of Neurospora crassa

Traits of the natural-death mutant of Neurospora crassa indicate that the nd+ allele codes for a function that is essential for stable maintenance of the mitochondrial chromosome, possibly a protein involved in replication, repair, or recombination.

Hyperactive recombination in the mitochondrial DNA of the natural death nuclear mutant of Neurospora crassa

The implication of these results is that the nd allele hyperactivates a general system for homologous recombination in the mitochondria of N. crassa.

Lipofuscin and its Relation to Aging

Phase Resetting of the Neurospora crassa Circadian Oscillator: Effects of Inositol Depletion on Sensitivity to Light

An indirect role for inositol metabolites in the light input pathway—one that is not likely to involve direct participation of an inositl phospholipid signal transduction mechanism is indicated.

Neurospora crassa . DNA of the natural death nuclear mutant of Hyperactive recombination in the mitochondrial

References

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Ageing of Neurospora crassa. I. Evidence for the free radical theory of ageing from studies of a natural-death mutant

Ageing of Neurospora crassa. IV. Induction of senescence in wild type by dietary amino acid analogs and reversal by antioxidants and membrane stabilizers

Inositol Deficiency Resulting in Death: An Explanation of Its Occurrence in Neurospora crassa

In mycelia of the mutant requiring inositol, grown in a suboptimum amount of exogenous inositols, the strlctural initegrity of the protease particles is lost, and the bulk of intracellular protease activity is recovered in the soluble fraction of the cell.

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Increase in microviscosity with ageing in protoplast plasmalemma of rose petals

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Glucose Utilization and Ethanolic Fermentation by Wild Type and Extrachromosomal Mutants of Neurospora crassa

It is suggested that the Pasteur effect, the inhibition of fermentation by respiration, may be operative in N. crassa and factors such as nitrogen source and concentration and oxygen tension, which may serve primarily to regulate the amount and form of respiration would, therefore, indirectly regulate fermentation.

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