Acetylcholine receptor agonists stimulate [3H]taurine release from rat sympathetic ganglia.

@article{Waniewski1994AcetylcholineRA,
  title={Acetylcholine receptor agonists stimulate [3H]taurine release from rat sympathetic ganglia.},
  author={Robert A. Waniewski and D. L. Martin},
  journal={European journal of pharmacology},
  year={1994},
  volume={260 2-3},
  pages={
          113-20
        }
}
3 Citations

Nicotinic receptors mediate the release of amino acid neurotransmitters in cultured cortical neurons.

Nicotine stimulation of cortical neurons obtained from gestation day 19 rats provoked a dose-dependent release of aspartate, glutamate, glycine and GABA, indicating a functional role for the nicotinic receptor in this model, and it was shown that L, N, and P/Q type Ca2+ channels are involved in nicotine-mediatedCa2+ entry into cortical neurons.

References

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The results suggest that taurine may act as a modulator of neuronal activity, possibly by stabilizing excitable membrane and by suppressing the release of neurotansmitter at synapses.

[3H]gamma‐Aminobutyric acid uptake into neuroglial cells of rat superior cervical sympathetic ganglia.

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Tachykinin‐Stimulated Inositol Phospholipid Hydrolysis and Taurine Release from Human Astrocytoma Cells

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