A new paraclinical CSF marker for hypoxia-like tissue damage in multiple sclerosis lesions.

Abstract

Recent studies on the immunopathology of multiple sclerosis revealed a heterogeneity in the patterns of demyelination, suggesting interindividual differences in the mechanism responsible for myelin destruction. One of these patterns of demyelination, characterized by oligodendrocyte dystrophy and apoptosis, closely mimics myelin destruction in acute white… (More)

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Cite this paper

@article{Lassmann2003ANP, title={A new paraclinical CSF marker for hypoxia-like tissue damage in multiple sclerosis lesions.}, author={Hans Lassmann and Markus Reindl and Helmut Rauschka and Johannes Berger and Fahmy Aboul-Enein and Thomas Berger and Andreas Zurbriggen and Andreas Lutterotti and Wolfgang Br{\"{u}ck and Joerg R. Weber and Robert Ullrich and Manfred Schmidbauer and Kurt A. Jellinger and Marc Vandevelde}, journal={Brain : a journal of neurology}, year={2003}, volume={126 Pt 6}, pages={1347-57} }