A mathematical framework for separating the direct and bystander components of cellular radiation response.

Abstract

UNLABELLED A mathematical model for fractional tumor cell survival was developed incorporating components of cell killing due to direct radiation interactions and bystander signals resulting from non-local dose deposition. MATERIAL AND METHODS Three possible mechanisms for signal production were tested by fitting predictions to available experimental… (More)
DOI: 10.3109/0284186X.2010.487874

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