A critical role for thiol, but not ATP, depletion in 95% O2-mediated injury of preterm pneumocytes in vitro.


Prolonged exposure to elevated partial pressures of oxygen results in lung cell toxicity, both in vivo and in vitro, due to the excess production and target molecule reactions of reactive oxygen species. Events primarily responsible for cell death vary with the type of oxidant injury and with cell type. Because of the susceptibility of the premature lung to… (More)


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