A cell-permeant peptide corresponding to the cUBP domain of USP5 reverses inflammatory and neuropathic pain

Abstract

BACKGROUND Cav3.2 T-type calcium currents in primary afferents are enhanced in various painful pathological conditions, whereas inhibiting Cav3.2 activity or expression offers a strategy for combating the development of pain hypersensitivity. We have shown that Cav3.2 channel surface density is strongly regulated by the ubiquitination machinery and we… (More)
DOI: 10.1177/1744806916642444

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Cite this paper

@inproceedings{GarcaCaballero2016ACP, title={A cell-permeant peptide corresponding to the cUBP domain of USP5 reverses inflammatory and neuropathic pain}, author={Agustin Garc{\'i}a-Caballero and Vinicius M. Gadotti and Lina Chen and Gerald W Zamponi}, booktitle={Molecular pain}, year={2016} }