3alpha,5alpha-Tetrahydroprogesterone (allopregnanolone) and gamma-aminobutyric acid: autocrine/paracrine interactions in the control of neonatal PSA-NCAM+ progenitor proliferation.

@article{Gago20043alpha5alphaTetrahydroprogesteroneA,
  title={3alpha,5alpha-Tetrahydroprogesterone (allopregnanolone) and gamma-aminobutyric acid: autocrine/paracrine interactions in the control of neonatal PSA-NCAM+ progenitor proliferation.},
  author={Nathalie Gago and Martine El-Etr and Nicole Sanan{\`e}s and Françoise Cadepond and Denise Samuel and Virginia Avellana-Adalid and Anne Baron-Van Evercooren and Michael Schumacher},
  journal={Journal of neuroscience research},
  year={2004},
  volume={78 6},
  pages={770-83}
}
The earliest identified neonatal neural progenitors are cells that express the polysialylated form of the neural cell adhesion molecule (PSA-NCAM). One of these progenitors is the early PSA-NCAM+ progenitor (ePSA-NCAM+ progenitor; Gago et al. [2003] Mol Cell Neurosci 22:162-178), which corresponds to a multipotential cell with a default differentiation through glial lineages. The ePSA-NCAM+ progenitor can synthesize the neurosteroid progesterone (PROG) and its reduced metabolite 3alpha,5alpha… CONTINUE READING
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