211 G to A Variation of UDP-Glucuronosyl Transferase 1A1 Gene and Neonatal Breastfeeding Jaundice

@article{Chou2011211GT,
  title={211 G to A Variation of UDP-Glucuronosyl Transferase 1A1 Gene and Neonatal Breastfeeding Jaundice},
  author={H. C. Chou and Mei Chen and H. Yang and Y. Su and W. Hsieh and C. Chen and Huey-Ling Chen and Mei-Hwei Chang and Po-Nien Tsao},
  journal={Pediatric Research},
  year={2011},
  volume={69},
  pages={170-174}
}
  • H. C. Chou, Mei Chen, +6 authors Po-Nien Tsao
  • Published 2011
  • Medicine
  • Pediatric Research
  • Breastfeeding jaundice is a common problem in neonates who were exclusively breastfed, but its pathogenesis is still unclear. The uridine diphosphate glucuronosyl transferase 1A1 (UGT1A1) gene polymorphism was shown to contribute to the development of neonatal hyperbilirubinemia. We hypothesize that the variation of UGT1A1 gene may contribute to neonatal breastfeeding jaundice. We prospectively enrolled 688 near-term and term infants who were exclusively breastfed (BF group) or were… CONTINUE READING
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    References

    SHOWING 1-10 OF 38 REFERENCES
    Molecular genetics of unconjugated hyperbilirubinemia in Taiwanese.
    • 19
    Gilbert syndrome accelerates development of neonatal jaundice.
    • 151
    Gilbert syndrome and glucose-6-phosphate dehydrogenase deficiency: a dose-dependent genetic interaction crucial to neonatal hyperbilirubinemia.
    • 303
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