17 beta-estradiol regulation of human endothelial cell basal nitric oxide release, independent of cytosolic Ca2+ mobilization.

@article{CaulinGlaser199717BR,
  title={17 beta-estradiol regulation of human endothelial cell basal nitric oxide release, independent of cytosolic Ca2+ mobilization.},
  author={Teresa Caulin-Glaser and Guillermo Garc{\'i}a-Carde{\~n}a and Phillip M. Sarrel and William C Sessa and Jeffrey R Bender},
  journal={Circulation research},
  year={1997},
  volume={81 5},
  pages={
          885-92
        }
}
Estradiol retards the development of atherosclerosis. Animal models have suggested that NO may be a critical effector molecule in this cardiovascular protection. In this study, female human umbilical vein endothelial cells (HUVECs) were propagated in phenol red-free gonadal hormone-free medium and pretreated with 17 beta-estradiol (E2). Reduced NO2- and NO3- (NOx) concentration, determined by chemiluminescence, demonstrated a rapid increase in basal HUVEC NO release in response to physiological… CONTINUE READING

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