• Corpus ID: 38959217

17 Depressive Disorders and Thyroid Function

@inproceedings{Araya201217DD,
  title={17 Depressive Disorders and Thyroid Function},
  author={A Ver{\'o}nica Araya and Teresa Massardo and Jenny L. Fiedler and Luis Risco and Juan C. Quintana and Claudio Liberman},
  year={2012}
}
A. Verónica Araya1, Teresa Massardo2, Jenny Fiedler3, Luis Risco4, Juan C. Quintana5 and Claudio Liberman1 1Endocrinology Section, Clinical Hospital of the University of Chile 2Nuclear Medicine Section, Clinical Hospital of the University of Chile 3Faculty of Chemical and Pharmaceutical Sciences, University of Chile 4Psychiatric Clinic of the Clinical Hospital of the University of Chile 5Department of Radiology, School of Medicine, Pontificia Universidad Católica de Chile Chile 

Figures from this paper

References

SHOWING 1-10 OF 109 REFERENCES

Thyroid-hormone enhancement of imipramine in nonretarded depressions.

The speed and efficacy of imipramine in the treatment of clinical depression was enhanced by the addition of tri-iodothyronine to the treatment program and physiologic changes were slight and within the limits of euthyroidism as usually defined.

Enhancement of imipramine antidepressant activity by thyroid hormone.

Twenty euthyroid patients with retarded depression were studied to determine the possible role of alterations in thyroid function in the etiology and treatment of depression, and T3 was physiologically active, altering protein-bound iodine values and accelerating ankle reflex time.

Potentiation of amitriptyline by thyroid hormone.

  • D. Wheatley
  • Medicine, Psychology
    Archives of general psychiatry
  • 1972
A double-blind comparison was made between amitriptyline alone and amitriptyline with added L-triiodothyronine (T3, liothyronine sodium at two dose levels, in cases of depression seen in general

CSF somatostatin in affective illness.

A significant inverse correlation was observed between somatostatin and the duration of sleep on the night of the lumbar puncture and 5-hydroxyindoleacetic acid and norepinephrine in the CSF.

Treatment of Refractory Depression with High-Dose Thyroxine

Thyroid and adrenal axis in major depression: a controlled study in outpatients.

When compared with matched control subjects, outpatients with major depression had slightly higher serum TSH, while urinary cortisol levels were similar, and it was observed lower serum cortisol in atypical depression than in non-atypical Depression.

T3 augmentation of SSRI resistant depression.

Thyrotropin, but not a polymorphism in type II deiodinase, predicts response to paroxetine in major depression.

Higher serum TSH was associated with response to paroxetine in patients with major depression, and response rates per tertile of HPT-axis parameters and per DII genotype significantly predicted response.

Thyroid dysfunction and affective illness.

Lithium treatment induces the formation of antibodies in susceptible people, which may lead to the development of thyroid disease in these patients, which is not significantly high when compared with the incidence in the general population.
...