0 Effects of He - 0 2 breathing during experimental decompression sickness following air dives

Abstract

Catron PW. Th as LB. Flynn ET Jr. Mcl)ermott J . Holt MA. Effects of He-() breathing during experi Idecompression sickness foil air dives. Undersea Biomed Res 1987: 14(2): 101II.he effects of ventilation with e2 during decompression sickness (DCS1 and venous air embolism were studied. Fifteen anestht zed dogs were mechanically ventilated and subjected to repeated air dives until pulmonary artery pressure at least doubled within 10 min postdive. At 30 .in postdive, ventilation was either continued with air (controls. it = 7) or changed to He-O19. n = 8t for an additional 90 rmin. All animals developed pulmonary hypertension. systemic hypotension. hemoconcentration. hypoxemia. hypercarbia. and pul monary edema. Breathing air or He-OWpostdive did not alter these responses, but He-OJ breathing produced an I I'/( increase in pulmonary vascular resistance IPVR). In 3 other anesthetized dogs that were not subjected to dives, ventilation was changed to He-O%' at various times during an intravenous infusion of air: He-OWbreathing caused a 22/ increase in PVR. We conclude that breathinHe-(nXuring DCS resulting from an air dive can intensify pulmonary vascular obstruction. l -'tr V i" " F ,

3 Figures and Tables

Cite this paper

@inproceedings{Catron0EO, title={0 Effects of He - 0 2 breathing during experimental decompression sickness following air dives}, author={Philip W. Catron and L Brannon Thomas and Ethan Flynn and John J. McDermott and Holt} }