[Course of acute oxidative damage after traumatic brain injury: problem and strategy].


During secondary pathophysiological cascades of early brain injury (EBI) after traumatic brain injury (TBI), reactive oxygen species (ROS) act rapidly play a pivotal role in the pathogenesis, neurodegeneration and prognosis of EBI via damaging lipid, protein and nucleic acid. However, the time spot of biological sample collection of oxidation-stress in TBI… (More)
DOI: 10.11817/j.issn.1672-7347.2014.02.015