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Toxicity and translocation of graphene oxide in Arabidopsis thaliana.
TLDR
The data imply that although GO was absorbed by Arabidopsis plants through root hairs, plants might still have strong ability to be against GO translocation into stem or leaves, and cotyledon might serve as an important site for GO distribution during the early development. Expand
Quantum dots exposure alters both development and function of D-type GABAergic motor neurons in nematode Caenorhabditis elegans
TLDR
This work examined the in vivo quantum dots neurotoxicity and the underlying mechanism using Caenorhabditis elegans D-type GABAergic motor neurons as the assay system and hypothesized that both translocation into the targeted neurons and alterations in the development and function of those targeted neurons contribute to the induction of CdTe QDs neurotoxicity. Expand
Transgenerational effects of traffic-related fine particulate matter (PM₂.₅) on nematode Caenorhabditis elegans.
TLDR
The data here imply the potential toxic effects of long-term exposure to traffic-related PM2.5 on environmental organisms, and further highlight the possible crucial roles of biological barrier and defecation behavior in regulating the PM 2.5 toxicity. Expand
Adverse Effects from Clenbuterol and Ractopamine on Nematode Caenorhabditis elegans and the Underlying Mechanism
TLDR
The Caenorhabditis elegans assay system may be useful for assessing the possible toxicity from weight loss agents, and clentuberol and ractopamine may induce toxicity through different molecular mechanisms. Expand
Response of microRNAs to in vitro treatment with graphene oxide.
TLDR
It is hypothesized that the dysregulated miRNAs could activate both a death receptor pathway by influencing functions of tumor necrosis factor α receptor and caspase-3 and a mitochondrial pathway by affecting functions of p53 and Bcl-2 in GO-exposed GLC-82 cells. Expand
Translocation and neurotoxicity of CdTe quantum dots in RMEs motor neurons in nematode Caenorhabditis elegans.
TLDR
The combinational effects of oxidative stress, cell identity, and bioavailability may contribute greatly to the mechanism of CdTe QDs neurotoxicity on RMEs motor neurons. Expand
microRNAs control of in vivo toxicity from graphene oxide in Caenorhabditis elegans.
TLDR
The authors report that multiple important pathways are influenced by GO and raise a hypothesis that GO may reduce lifespan through influencing the functions of insulin/IGF signaling, TOR signaling, and germline signaling pathways controlled by miRNAs. Expand
p38 MAPK-SKN-1/Nrf signaling cascade is required for intestinal barrier against graphene oxide toxicity in Caenorhabditis elegans
TLDR
In vivo assay system of Caenorhabditis elegans found that mutation of genes encoding core p38 mitogen-activated protein kinase (MAPK) signaling pathway caused susceptible property to GO toxicity and enhanced translocation of GO into the body of nematodes, suggesting that p38 MAPK-SKN-1/Nrf signaling cascade may act as an important molecular basis for intestinal barrier against GO toxicity in organisms. Expand
Simultaneous qualitative and quantitative analysis of 21 mycotoxins in Radix Paeoniae Alba by ultra-high performance liquid chromatography quadrupole linear ion trap mass spectrometry and QuEChERS
TLDR
The method proposed herein with significant advantages including simple pretreatment, rapid determination as well as high sensitivity, accuracy and throughput would be a preferred candidate for the determination and quantification of multi-class mycotoxin contaminants in real samples. Expand
Biosafety assessment of titanium dioxide nanoparticles in acutely exposed nematode Caenorhabditis elegans with mutations of genes required for oxidative stress or stress response.
TLDR
The results suggest the relatively safe property of acute exposure to TiO2-NPs with certain durations at predicted environmental relevant concentrations or concentrations comparable to those in food in nematodes with mutations of some susceptible genes. Expand
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