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BACKGROUND Hepatitis B virus (HBV) infection has a tendency to be chronic. The quasi species of HBV in the pre-S/S gene and mutations in enhancer II (EnhII)/core promoter (CP)/pre-C of HBV were(More)
OBJECTIVE To understand the relationship between the different replication status of HBV and mutations in core promoter in mother and child infected by mother-to-infant transmission. METHODS Core(More)
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