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  • Yuko Atsumi, Aki Inase, +10 authors Ken-ichi Yoshioka
  • Biology, Medicine
  • The Journal of Biological Chemistry
  • 2013 (First Publication: 27 March 2013)
  • Background: It is unclear how DNA-damaging agents target cancer cells over normal somatic cells. Results: Arf/p53-dependent down-regulation of H2AX enables normal cells to survive after DNA damage.Expand
  • Yusuke Minakawa, Yuko Atsumi, Akira Shinohara, Yasufumi Murakami, Ken-ichi Yoshioka
  • Chemistry, Biology, Medicine
  • Genes to cells : devoted to molecular & cellular…
  • 2016 (First Publication: 1 July 2016)
  • H2AX is expressed at very low levels in quiescent normal cells in vivo and in vitro. Such cells repair DNA double-strand breaks (DSBs) induced by γ-irradiation through a transient stabilization ofExpand
  • Tomoyuki Osawa, Yuko Atsumi, +5 authors Ken-ichi Yoshioka
  • Medicine, Biology
  • Biochemical and biophysical research…
  • 2013 (First Publication: 1 March 2013)
  • Normal cells undergo a growth-arrested status that is produced by p53-dependent down-regulation of histone H2AX. Immortality is developed after abrogation of the H2AX-diminished state, which isExpand