Share This Author
Synergistic antibacterial effect of curcumin against methicillin-resistant Staphylococcus aureus.
Alantolactone suppresses inducible nitric oxide synthase and cyclooxygenase-2 expression by down-regulating NF-κB, MAPK and AP-1 via the MyD88 signaling pathway in LPS-activated RAW 264.7 cells.
Cytotoxic components from the dried rhizomes of Zingiber officinaleRoscoe
Five compounds were isolated from the chloroform-soluble fraction of the methanolic extract of the dried rhizomes of Zingiber officinale (Zingiberaceae) through repeated column chromatography and 6-shogaol exhibited the most potent cytotoxicity against human A549,SK-OV-3, SK-MEL-2, and HCT15 tumor cells.
The Neoflavonoid Latifolin Isolated from MeOH Extract of Dalbergia odorifera Attenuates Inflammatory Responses by Inhibiting NF‐κB Activation via Nrf2‐Mediated Heme Oxygenase‐1 Expression
Results suggested that the latifolin‐mediated up‐regulation of HO‐1 expression played a critical role in anti‐inflammatory effects in macrophages.
Hepatoprotective and free radical scavenging activities of phenolic petrosins and flavonoids isolated from Equisetum arvense.
Enhanced paclitaxel bioavailability after oral administration of paclitaxel or prodrug to rats pretreated with quercetin.
Curcumin induces pro-apoptotic endoplasmic reticulum stress in human leukemia HL-60 cells.
Scopoletin suppresses pro-inflammatory cytokines and PGE2 from LPS-stimulated cell line, RAW 264.7 cells.
Roles of heme oxygenase-1 in curcumin-induced growth inhibition in rat smooth muscle cells
It is suggested that curcumin has an ability to induce HO-1 expression, presumably through Nrf2-dependent ARE activation, in rat VSMCs and HASMCs, and evidence that the antiproliferative effect ofCurcumin is considerably linked to its ability to induced HO- 1 expression is provided.
Comparative effects of curcuminoids on endothelial heme oxygenase-1 expression: ortho-methoxy groups are essential to enhance heme oxygenase activity and protection
Cytoprotective effects afforded by curcuminoids were considerably associated with their abilities to enhance HO activity, and may be useful in designing more efficacious HO-1 inducers which could be considered as promising pharmacological agents for the prevention or treatment of endothelial diseases caused by oxidative damages.