Yoshitaka Imai

Learn More
The cause of Leber’s disease probably lies in abnormal cytoplasm, inheritance being therefore maternal (Lossen’s law). The abnormal cytoplasm continues in the egg of the affected pedigrees from generation to generation, so that all females are carriers of the disease (Kitashima’s law). The disease, in its attack, is stimulated by a sex hormone, especially(More)
Introdnct~ion . 203 Expm'imentM ~n~lysls of flower colour 20~ Whi~e flowers 20~I :Flower hues 205 Flower gones 206 Flower pM~erns . 207 h ' fn~ing ch~ra, c6ers 21.0 :Flecked pedigree 211 Som'ce of m~mq~l a.nd inbreeding exI~el~ments 211 Whit~e v~l'iM;ion 213 Nolanal mu t,a.n~s 213 Mutating sta,ges 21~i :PericlhlM ~orms 2]5 Dnskish pe, dklgree 217(More)
1. The partial inhibitor, Fp, described in the present paper, is quite different in its behavior from Ff, discussed in a previous paper (1926). The factor Fp suppresses the complete development of the white margin in producing it in the “half”-“slight” degrees. 2. The dominance of Fp is almost perfect. 3. The manifestation of this partial inhibitor is(More)
AcCOaDIX-c~ to experiments conducted by Barker (1917), the flaked character of P/~'bit.is purpu~ea, the Norning Glory, behaves as simple dominant to the selLcoloured condition, presenting a heterozygous flower colour with intense anthoeyanin variegation. From the modern genetic point of view, however, the flaked condition is not so simple in its behaviour.(More)
J nl, l 'Oduotion ~J'ho "pa.rentrl,1 s t o c k " ~l'ypes of doub l i ng J>oi, a.loid doub lo f lowors The OOllllllOll l, ypo ':[~]lo " K u j t t k u " l,ype {l ' l io <~]Jf)|)llll >> f lower {l'ho <'([ 'onltga,-BolAul" I lowor The segregll, t,ing rM, io of l, ho "Sh ish i -Bo l~an" f lower Colnbin,~l~ioils o[ doub l i i l g fiu~l, ors Tho effcel, o f a l le(More)